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dc.contributor.authorShanklin, D. Radford
dc.date.accessioned2011-05-26T15:49:06Z
dc.date.available2011-05-26T15:49:06Z
dc.date.issued2011-04-10
dc.identifier.urihttp://hdl.handle.net/1912/4625
dc.descriptionAuthor Posting. © The Author(s), 2011. Presented at Advances in inflammatory and fibrosing lung diseases. Sunday, April 10, 2011, Experimental Biology 2011, Washington, D.C.en_US
dc.description.abstractPulmonary fibrosis implies antecedent lung injury which may or may not include inflammatory responses of the ordinary sort. The onset of breathing at mammalian birth is a different kind of lung injury, one occasioned by great physical stretch of the collapsed but moist fetal lung, and immediate exposure to over ten times the level of oxygen resident in the fetal organ. Access to a large archive, the perinatal mortality review from the Chicago Lying-In Hospital, has provided information very relevant to these questions, including the first regular documentation of the pulmonary lesion complex as related to clinical care: beginning in the late 1930s. The lesion complex is called hyaline membrane disease (HMD) from the condensation at the tissue:gas interface of protein exuded from the lung and its circulation.en_US
dc.format.mimetypeapplication/pdf
dc.language.isoen_USen_US
dc.titleThyroid and adrenal factors in hyaline membrane diseaseen_US
dc.typePresentationen_US


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